<P> Virus latency is not to be confused with clinical latency during the incubation period when a virus is not dormant . </P> <P> Episomal latency refers to the use of genetic episomes during latency . In this type, viral genes are stabilized floating in the cytoplasm or nucleus as distinct objects, either as linear or lariat structures . Episomal latency is more vulnerable to ribozymes or host foreign gene degradation than provirus latency . </P> <P> One example is herpes virus family, Herpesviridae, all of which establish latent infection . Herpes virus include chicken - pox virus and herpes simplex viruses (HSV - 1, HSV - 2), all of which establish episomal latency in neurons and leave linear genetic material floating in the cytoplasm . The Gammaherpesvirinae subfamily is associated with episomal latency established in cells of the immune system, such as B - cells in the case of Epstein - Barr virus . Epstein - Barr virus lytic reactivation (which can be due to chemotherapy or radiation) can result in genome instability and cancer . In the case of herpes simplex (HSV), the virus has been shown to fuse with DNA in neurons, such as nerve ganglia or brain cells, and HSV reactivates upon even minor chromatin loosening with stress, although the chromatin compacts (becomes latent) upon oxygen and nutrient deprivation . </P> <P> Cytomegalovirus (CMV) establishes latency in myeloid progenitor cells, and is reactivated by inflammation . Immunosuppression and critical illness (sepsis in particular) often results in CMV reactivation . CMV reactivation is commonly seen in patients with severe colitis . </P>

When the herpesvirus is latent it resides in which area
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