<Li> muscarinic agonists, e.g. acetylcholine → muscarinic receptor M </Li> <Li> NPY → NPY receptor </Li> <Tr> <Td> hepatocyte </Td> <Td> liver </Td> <Td> <Ul> <Li> epinephrine → β - adrenergic receptor </Li> <Li> glucagon → Glucagon receptor </Li> </Ul> </Td> <Td> </Td> <Td> <Ul> <Li> produce glucose <Ul> <Li> stimulate glycogenolysis <Ul> <Li> phosphorylate glycogen phosphorylase (activating it) </Li> <Li> phosphorylate Acetyl - CoA carboxylase (inhibiting it) </Li> </Ul> </Li> <Li> inhibit glycogenesis <Ul> <Li> phosphorylate glycogen synthase (inhibiting it) </Li> </Ul> </Li> <Li> stimulate gluconeogenesis <Ul> <Li> phosphorylate fructose 2, 6 - bisphosphatase (stimulating it) </Li> </Ul> </Li> <Li> inhibit glycolysis <Ul> <Li> phosphorylate phosphofructokinase - 2 (inactivating it) </Li> <Li> phosphorylate fructose 2, 6 - bisphosphatase (stimulate it) </Li> <Li> phosphorylate pyruvate kinase (inhibiting it) </Li> </Ul> </Li> </Ul> </Li> </Ul> </Td> </Tr> <Ul> <Li> epinephrine → β - adrenergic receptor </Li> <Li> glucagon → Glucagon receptor </Li> </Ul>

How does protein kinase a cause a sodium channel to open